Should we really stress about having a 'high' bad cholesterol level?
Raised cholesterol levels are noticed as a risk factor for cardiovascular system disease and statins are seen as the get rid of. I already knew that statins had side-effects so once i was told my bad cholesterol was too high I actually wanted to determine more.
What follows is not really a medical act, a professional view, or a qualified view, simply a summary of information available on the net which We personally found convincing.
UK doctors consider a hypercholesteria level over 5mml/l (millimoles per litre) to be 'high' and an signal of increased risk of heart attacks and strokes. In countries such as Switzerland and France the average cholesterol levels are higher than yet the incidence of heart problems and strokes is much below ours. In Russia the typical cholesterol levels are much lower and yet the incidence of heart disease is much higher than ours.
How can this be?
Cholesterol is a fatty acid created by the liver and used throughout the body to create new cells and create hormones. This is a natural and necessary function and without cholesterol we would die.
As extra fat is not soluble within our watery blood it must increase a protein 'carrier' to create molecules of low density lipoprotein (LDL) for delivery to where it is necessary. Unused bad cholesterol is then carried back again to the liver via high-density lipoproteins (HDL) for recycling.
There is no good or bad hypercholesteria, just cholesterol and even if we had a cholesterol-free diet our body would still be placing it up.
It is the physical size of the LDL particles which are or worry. If they are too small chances are they can get lodged between flakes of arterial plaque turning rancid over time to increase localised infection. To ensure that cholesterol to have this effect the arterial blood vessels must currently have plaque. Center disease isn't caused by cholesterol but if you are already susceptible then it can make things worse: so could any fat in the blood vessels such as omega-3 from oily fish!
We have to have more of the much larger LDL particles to reduce our risk and we can do this with becomes our diet and so avoid harmful medication. Interestingly it seems that alcohol and saturated fat in the diet actually will raise the size of BAD particles. Sound familiar? Certainly, just as the French diet!
The formation of plaque on arterial walls is in response to damaging harmful toxins carried by the blood vessels. Anything we ingest will sooner or later pass through the blood stream so could cause damage - including viruses and bacteria. Normally blood vessels will behave to damage by becoming inflamed - which neutralises the toxins - then the inflammation dies down and the repair process starts. This process requires a matter of hours and is happening all the time.
Sometimes, however, the inflammation persists but the repair process still will try to take a nap new skin cells. The inflammation will try to break down the new cells and it becomes a localised challenge. Even if the maintenance work, they are just covering inflamed areas (with plaque) and this can continue for several years. Eventually the loose plaque may be dislodged and cause destruction as it moves through the blood stream. The end result can be strokes or heart episodes.
Although cholesterol may make things worse - as any oil in the blood stream could -it isn't the root cause of the condition. It seems cholesterol is getting a poor press.
Statins reduce the overall amount of bad cholesterol nonetheless they also have many side-effects such as feeling giddy, loss of muscle-mass, storage problems and lower bone-mass. There is also a risk of kidney destruction. This is not amazing as statins are interfering with a natural and necessary process.
Some research indicates that statins can help prevent an additional cardiovascular attack but there is little evidence that taking statins makes any big difference to the risk of having the first myocardial infarction. It seems that statins do not have an effect on the size of particles - just the overall volume - so these results are no surprise.
The threshold of 'high' cholesterol is changing and arbitrary yet it can result in approved medication in the form of statins which can have serious side-effects. Having a high overall cholesterol level need not be bad media but taking statins practically always is. Statins are not addressing any main problems some individuals may have nevertheless they could be supplying false hope.
Raised cholesterol levels are noticed as a risk factor for cardiovascular system disease and statins are seen as the get rid of. I already knew that statins had side-effects so once i was told my bad cholesterol was too high I actually wanted to determine more.
UK doctors consider a hypercholesteria level over 5mml/l (millimoles per litre) to be 'high' and an signal of increased risk of heart attacks and strokes. In countries such as Switzerland and France the average cholesterol levels are higher than yet the incidence of heart problems and strokes is much below ours. In Russia the typical cholesterol levels are much lower and yet the incidence of heart disease is much higher than ours.
How can this be?
Cholesterol is a fatty acid created by the liver and used throughout the body to create new cells and create hormones. This is a natural and necessary function and without cholesterol we would die.
As extra fat is not soluble within our watery blood it must increase a protein 'carrier' to create molecules of low density lipoprotein (LDL) for delivery to where it is necessary. Unused bad cholesterol is then carried back again to the liver via high-density lipoproteins (HDL) for recycling.
There is no good or bad hypercholesteria, just cholesterol and even if we had a cholesterol-free diet our body would still be placing it up.
It is the physical size of the LDL particles which are or worry. If they are too small chances are they can get lodged between flakes of arterial plaque turning rancid over time to increase localised infection. To ensure that cholesterol to have this effect the arterial blood vessels must currently have plaque. Center disease isn't caused by cholesterol but if you are already susceptible then it can make things worse: so could any fat in the blood vessels such as omega-3 from oily fish!
We have to have more of the much larger LDL particles to reduce our risk and we can do this with becomes our diet and so avoid harmful medication. Interestingly it seems that alcohol and saturated fat in the diet actually will raise the size of BAD particles. Sound familiar? Certainly, just as the French diet!
The formation of plaque on arterial walls is in response to damaging harmful toxins carried by the blood vessels. Anything we ingest will sooner or later pass through the blood stream so could cause damage - including viruses and bacteria. Normally blood vessels will behave to damage by becoming inflamed - which neutralises the toxins - then the inflammation dies down and the repair process starts. This process requires a matter of hours and is happening all the time.
Sometimes, however, the inflammation persists but the repair process still will try to take a nap new skin cells. The inflammation will try to break down the new cells and it becomes a localised challenge. Even if the maintenance work, they are just covering inflamed areas (with plaque) and this can continue for several years. Eventually the loose plaque may be dislodged and cause destruction as it moves through the blood stream. The end result can be strokes or heart episodes.
Although cholesterol may make things worse - as any oil in the blood stream could -it isn't the root cause of the condition. It seems cholesterol is getting a poor press.
Statins reduce the overall amount of bad cholesterol nonetheless they also have many side-effects such as feeling giddy, loss of muscle-mass, storage problems and lower bone-mass. There is also a risk of kidney destruction. This is not amazing as statins are interfering with a natural and necessary process.
Some research indicates that statins can help prevent an additional cardiovascular attack but there is little evidence that taking statins makes any big difference to the risk of having the first myocardial infarction. It seems that statins do not have an effect on the size of particles - just the overall volume - so these results are no surprise.
The threshold of 'high' cholesterol is changing and arbitrary yet it can result in approved medication in the form of statins which can have serious side-effects. Having a high overall cholesterol level need not be bad media but taking statins practically always is. Statins are not addressing any main problems some individuals may have nevertheless they could be supplying false hope.
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